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Effexor Birth Injury Lawsuit Claims Fatal Respiratory Problem in Newborns
Effexor Birth Defects are a serious injury. A Tennessee mother gave birth to a boy with life-threatening persistent pulmonary hypertension (PPHN) as a result of antidepressant Effexor birth injury side effects. The pregnant woman had taken the Effexor antidepressant so that she could maintain a healthy emotional state and prevent any prenatal injury or suffering to the fetus because of her conditions. She believed the antidepressant was without any side effect and could be a safe solution for her mental condition.
However, the repercussions of her taking the medicine during pregnancy were extremely severe and unexpected. Effexor side effects resulted in the birth of the boy with PPHN, a serious birth defect that restricts the lung functions and prevents oxygen from reaching vital organs. The child was put on a neonatal intensive care unit respirator for a week, as he could not breathe properly for a few minutes after his birth and experienced life-threatening conditions.
According to the Effexor birth injury lawsuit filed by the aggrieved parents, PPHN caused by Effexor birth defect side effects resulted in near-fatal condition for their child and despite early treatment, the child continues to suffer health problems and needs constant monitoring for years. In addition, they blame the manufacturer for their emotional suffering and financial losses solely because it did not warn pregnant women about Effexor birth injury risks.
Effexor Birth Injury Side Effects and PPHN
Venlafaxine sold under the brand name Effexor by Pfizer is a serotonin-norepinephrine reuptake inhibitor, a class of more potent antidepressants that impacts enzymes controlling emotional state in our brain. A number of research reports have warned of potential danger of persistent pulmonary hypertension in children born to mothers treated with the antidepressant during pregnancy.
According to the New England Journal of Medicine, Effexor birth injury side effects may result in a six-fold rise in fatal PPHN risk if the antidepressant was consumed by pregnant women on or after 20th week of pregnancy. The study acted as catalyst for many to seek product liability against the manufacturer. The FDA and Health Canada also issued public advisories in the coming years, cautioning expectant mothers against ant-depressant use.
Effexor PPHN: The Fatal Condition
Persistent pulmonary hypertension caused by Effexor birth defects in newborns denotes a type of lung disorder that inhibits blood oxygen circulation and results in severe respiratory distress. Once the child is born, the lungs become active to supply oxygen to the body, which is performed through the neural tube during the prenatal stage. The incompetence of lungs due to PPHN birth defect disorder restricts pulmonary arteries entrusted with the function of ensuring smooth flow of blood oxygen. As a result, blood pressure shoots up to life-threatening level damaging vital body organs because of restricted blood flow and oxygen paucity.
Newborns with PPHN experience breathing problems and enhanced heart rate. Researchers have found PPHN in newborns is primarily because of the inability of the blood vessel, which carries prenatal oxygen supply to babies, to shut properly. This stops blood from reaching lungs, which, in turn, fail to carry adequate oxygen to different parts of the human body. Effexor antidepressant side effects during prenatal stage play a role in resulting in a congenital injury.
The condition may lead to heart and kidney failures and seizures if it is not treated immediately and successfully. Persistent pulmonary hypertension in newborns has resulted in a large number of Effexor birth defect lawsuits filed across the federal courts in the United States.
If you or a loved one took Effexor and a child was born with birth defects,please fill out the form to the right or call us so that we can provide you with forms to evaluate your potential case immediately. You may also visit our Effexor Birth Defect Lawsuit Information Source.
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